How (and why) to Ramble on your goat sideways
07-30-2011, 09:38 PM
#9083
I've spoken to some of my other friends in the Air Force and they all tell me the same thing, "JOIN THE AIR FORCE!!!!!!" But then their reasons are really, really lame. "I do nothing, lots of time off and I barely have to work." That would absolutely drive me crazy. I want to work and stay very busy.
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07-30-2011, 09:48 PM
#9084
Prepare your anus for a large PM
I'm going to post a **** load of cadences in the random picture thread, push you over that fence.
I've spoken to some of my other friends in the Air Force and they all tell me the same thing, "JOIN THE AIR FORCE!!!!!!" But then their reasons are really, really lame. "I do nothing, lots of time off and I barely have to work." That would absolutely drive me crazy. I want to work and stay very busy.
*According to the aforementioned book.
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07-30-2011, 09:59 PM
#9085
I'm a terrible person
iTrader: (19)
Join Date: Apr 2009
Location: Arizona
Posts: 7,174
Total Cats: 180
I actually retract my generalization to say that the Marines I have met have 9 times out of 10 been all show. Which seeing as I help train them, is a lot. There are always exceptions. I have respect for every service (even the AF). I have a lot of friends who are Marines/AF/Army etc.. Everyone has their purpose.
OP, you really need to figure out what you want to do. ASVAB isn't **** once you score above 90. Hell I haven't even met anyone that has scored below that in my MOS. Most max it.
I guess the big question is do you see yourself in the military for a career, or as a stepping stone?
OP, you really need to figure out what you want to do. ASVAB isn't **** once you score above 90. Hell I haven't even met anyone that has scored below that in my MOS. Most max it.
I guess the big question is do you see yourself in the military for a career, or as a stepping stone?
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07-30-2011, 10:16 PM
#9086
As for OP, if your thinking of joining either one as a career then there is the fact that the Army is bigger and therefor has more room to rise in the ranks, but that's not to say you couldn't do well in the MC, with your college you should get a promotion to PFC, or go to OCS if you want. I believe (not sure, you'd have to ask your recruiter) your DI's can promote you as well, so if you already have college you could (if you work really hard in boot camp) get promoted to Lance Corporal (again, I'm not at all sure of this, I've only done a little research and my sources aren't that great).
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07-30-2011, 11:04 PM
#9087
I'm a terrible person
iTrader: (19)
Join Date: Apr 2009
Location: Arizona
Posts: 7,174
Total Cats: 180
You should be excited. I have a lot of respect for Marines. But don't discount the Soldiers
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07-30-2011, 11:25 PM
#9088
*IF YOU HAVE NOT WATCHED THAT MOVIE, DO IT RIGHT NOW*
This is what happens when you don't salute a Officer
This is what happens when you say "need"
You should read 'Making the Coprs', by Thomas Ricks It's about going through boot camp and the history (good and bad) of the Corps.
*According to the aforementioned book.
*According to the aforementioned book.
As for OP, if your thinking of joining either one as a career then there is the fact that the Army is bigger and therefor has more room to rise in the ranks, but that's not to say you couldn't do well in the MC, with your college you should get a promotion to PFC, or go to OCS if you want. I believe (not sure, you'd have to ask your recruiter) your DI's can promote you as well, so if you already have college you could (if you work really hard in boot camp) get promoted to Lance Corporal (again, I'm not at all sure of this, I've only done a little research and my sources aren't that great).
My resources haven't been great either. Like I said, my Marine recruiter is a bit useless(so far). The forums are GREAT, though. If you're not one yet, I suggest you do that immediately.
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07-30-2011, 11:43 PM
#9089
I've seen Ears Open Eyeballs Click a few times, really good movie.
It sucks that your recruiter isn't as good as he should be, I can see how it certainly makes your decisions harder. I've been very lucky that all the recruiters at my RSS (and the Sargent Major who occasionally stops by) are cool guys who eat/breath/sleep Marine Corps. If you have a question that your recruiter can't answer or something like that, you may want to call 1-800-Marines and maybe you can get a better answer there.
It sucks that your recruiter isn't as good as he should be, I can see how it certainly makes your decisions harder. I've been very lucky that all the recruiters at my RSS (and the Sargent Major who occasionally stops by) are cool guys who eat/breath/sleep Marine Corps. If you have a question that your recruiter can't answer or something like that, you may want to call 1-800-Marines and maybe you can get a better answer there.
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07-31-2011, 12:30 AM
#9090
You don't need anything but air, water and 8 counts recruit.
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07-31-2011, 03:21 AM
#9091
Elite Member
iTrader: (1)
Join Date: Feb 2008
Location: Birmingham Alabama
Posts: 7,930
Total Cats: 45
How is it possible to be apathetic and anxious at the same time? 
I've been finding myself both of these for the last month or two, either alternately or at the same time. Apathy being far more predominant.
Some people say I'm apathetic, but I really don't care.
I've been finding myself both of these for the last month or two, either alternately or at the same time. Apathy being far more predominant. Some people say I'm apathetic, but I really don't care.
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07-31-2011, 10:18 AM
#9093
Boost Pope
iTrader: (8)
Join Date: Sep 2005
Location: Chicago. (The less-murder part.)
Posts: 33,038
Total Cats: 6,604
Genetic
Genetic studies have suggested many chromosomal regions and candidate genes appearing to relate to the development of bipolar disorder, but the results are not consistent and often not replicated.[28]
Although the first genetic linkage finding for mania was in 1969,[29] the linkage studies have been inconsistent.[30] Meta-analyses of linkage studies detected either no significant genome-wide findings or, using a different methodology, only two genome-wide significant peaks, on chromosome 6q and on 8q21.[citation needed] Genome-wide association studies neither brought a consistent focus — each has identified new loci.[30]
Findings point strongly to heterogeneity, with different genes being implicated in different families.[31] A review seeking to identify the more consistent findings suggested several genes related to serotonin (SLC6A4 and TPH2), dopamine (DRD4 and SLC6A3), glutamate (DAOA and DTNBP1), and cell growth and/or maintenance pathways (NRG1, DISC1 and BDNF), although noting a high risk of false positives in the published literature. It was also suggested that individual genes are likely to have only a small effect and to be involved in some aspect related to the disorder (and a broad range of "normal" human behavior) rather than the disorder per se.[32]
Advanced paternal age has been linked to a somewhat increased chance of bipolar disorder in offspring, consistent with a hypothesis of increased new genetic mutations.[33]
Physiological
Abnormalities in the structure and/or function of certain brain circuits could underlie bipolar. Two meta-analyses of MRI studies in bipolar disorder report a increase in the volume of the lateral ventricles, globus pallidus and increase in the rates of deep white matter hyperintensities.[34][35]
The "kindling" theory asserts that people who are genetically predisposed toward bipolar disorder can experience a series of stressful events,[36] each of which lowers the threshold at which mood changes occur. Eventually, a mood episode can start (and become recurrent) by itself. There is evidence of hypothalamic-pituitary-adrenal axis (HPA axis) abnormalities in bipolar disorder due to stress.[37]
Other brain components which have been proposed to play a role are the mitochondria,[38] and a sodium ATPase pump,[39] causing cyclical periods of poor neuron firing (depression) and hypersensitive neuron firing (mania). This may only apply for type one, but type two apparently results from a large confluence of factors.[citation needed] Circadian rhythms and melatonin activity also seem to be altered.[40]
Environmental
Evidence suggests that environmental factors play a significant role in the development and course of bipolar disorder, and that individual psychosocial variables may interact with genetic dispositions.[32] There is fairly consistent evidence from prospective studies that recent life events and interpersonal relationships contribute to the likelihood of onsets and recurrences of bipolar mood episodes, as they do for onsets and recurrences of unipolar depression.[41] There have been repeated findings that between a third and a half of adults diagnosed with bipolar disorder report traumatic/abusive experiences in childhood, which is associated on average with earlier onset, a worse course, and more co-occurring disorders such as PTSD.[42] The total number of reported stressful events in childhood is higher in those with an adult diagnosis of bipolar spectrum disorder compared to those without, particularly events stemming from a harsh environment rather than from the child's own behavior.[43] Early experiences of adversity and conflict are likely to make subsequent developmental challenges in adolescence more difficult, and are likely a potentiating factor in those at risk of developing bipolar disorder.[44]
http://en.wikipedia.org/wiki/Bipolar_disorder
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07-31-2011, 11:03 AM
#9094
Elite Member
iTrader: (1)
Join Date: Feb 2008
Location: Birmingham Alabama
Posts: 7,930
Total Cats: 45
The causes of bipolar disorder likely vary between individuals. Twin studies have been limited by relatively small sample sizes but have indicated a substantial genetic contribution, as well as environmental influence. For bipolar I, the (probandwise) concordance rates in modern studies have been consistently put at around 40% in monozygotic twins (same genes), compared to 0 to 10% in dizygotic twins.[25] A combination of bipolar I, II and cyclothymia produced concordance rates of 42% vs 11%, with a relatively lower ratio for bipolar II that likely reflects heterogeneity. The overall heritability of the bipolar spectrum has been put at 0.71.[26] There is overlap with unipolar depression and if this is also counted in the co-twin the concordance with bipolar disorder rises to 67% in monozigotic twins and 19% in dizigotic.[27] The relatively low concordance between dizygotic twins brought up together suggests that shared family environmental effects are limited, although the ability to detect them has been limited by small sample sizes.[26]
Genetic
Genetic studies have suggested many chromosomal regions and candidate genes appearing to relate to the development of bipolar disorder, but the results are not consistent and often not replicated.[28]
Although the first genetic linkage finding for mania was in 1969,[29] the linkage studies have been inconsistent.[30] Meta-analyses of linkage studies detected either no significant genome-wide findings or, using a different methodology, only two genome-wide significant peaks, on chromosome 6q and on 8q21.[citation needed] Genome-wide association studies neither brought a consistent focus — each has identified new loci.[30]
Findings point strongly to heterogeneity, with different genes being implicated in different families.[31] A review seeking to identify the more consistent findings suggested several genes related to serotonin (SLC6A4 and TPH2), dopamine (DRD4 and SLC6A3), glutamate (DAOA and DTNBP1), and cell growth and/or maintenance pathways (NRG1, DISC1 and BDNF), although noting a high risk of false positives in the published literature. It was also suggested that individual genes are likely to have only a small effect and to be involved in some aspect related to the disorder (and a broad range of "normal" human behavior) rather than the disorder per se.[32]
Advanced paternal age has been linked to a somewhat increased chance of bipolar disorder in offspring, consistent with a hypothesis of increased new genetic mutations.[33]
Physiological
Abnormalities in the structure and/or function of certain brain circuits could underlie bipolar. Two meta-analyses of MRI studies in bipolar disorder report a increase in the volume of the lateral ventricles, globus pallidus and increase in the rates of deep white matter hyperintensities.[34][35]
The "kindling" theory asserts that people who are genetically predisposed toward bipolar disorder can experience a series of stressful events,[36] each of which lowers the threshold at which mood changes occur. Eventually, a mood episode can start (and become recurrent) by itself. There is evidence of hypothalamic-pituitary-adrenal axis (HPA axis) abnormalities in bipolar disorder due to stress.[37]
Other brain components which have been proposed to play a role are the mitochondria,[38] and a sodium ATPase pump,[39] causing cyclical periods of poor neuron firing (depression) and hypersensitive neuron firing (mania). This may only apply for type one, but type two apparently results from a large confluence of factors.[citation needed] Circadian rhythms and melatonin activity also seem to be altered.[40]
Environmental
Evidence suggests that environmental factors play a significant role in the development and course of bipolar disorder, and that individual psychosocial variables may interact with genetic dispositions.[32] There is fairly consistent evidence from prospective studies that recent life events and interpersonal relationships contribute to the likelihood of onsets and recurrences of bipolar mood episodes, as they do for onsets and recurrences of unipolar depression.[41] There have been repeated findings that between a third and a half of adults diagnosed with bipolar disorder report traumatic/abusive experiences in childhood, which is associated on average with earlier onset, a worse course, and more co-occurring disorders such as PTSD.[42] The total number of reported stressful events in childhood is higher in those with an adult diagnosis of bipolar spectrum disorder compared to those without, particularly events stemming from a harsh environment rather than from the child's own behavior.[43] Early experiences of adversity and conflict are likely to make subsequent developmental challenges in adolescence more difficult, and are likely a potentiating factor in those at risk of developing bipolar disorder.[44]
http://en.wikipedia.org/wiki/Bipolar_disorder
Genetic
Genetic studies have suggested many chromosomal regions and candidate genes appearing to relate to the development of bipolar disorder, but the results are not consistent and often not replicated.[28]
Although the first genetic linkage finding for mania was in 1969,[29] the linkage studies have been inconsistent.[30] Meta-analyses of linkage studies detected either no significant genome-wide findings or, using a different methodology, only two genome-wide significant peaks, on chromosome 6q and on 8q21.[citation needed] Genome-wide association studies neither brought a consistent focus — each has identified new loci.[30]
Findings point strongly to heterogeneity, with different genes being implicated in different families.[31] A review seeking to identify the more consistent findings suggested several genes related to serotonin (SLC6A4 and TPH2), dopamine (DRD4 and SLC6A3), glutamate (DAOA and DTNBP1), and cell growth and/or maintenance pathways (NRG1, DISC1 and BDNF), although noting a high risk of false positives in the published literature. It was also suggested that individual genes are likely to have only a small effect and to be involved in some aspect related to the disorder (and a broad range of "normal" human behavior) rather than the disorder per se.[32]
Advanced paternal age has been linked to a somewhat increased chance of bipolar disorder in offspring, consistent with a hypothesis of increased new genetic mutations.[33]
Physiological
Abnormalities in the structure and/or function of certain brain circuits could underlie bipolar. Two meta-analyses of MRI studies in bipolar disorder report a increase in the volume of the lateral ventricles, globus pallidus and increase in the rates of deep white matter hyperintensities.[34][35]
The "kindling" theory asserts that people who are genetically predisposed toward bipolar disorder can experience a series of stressful events,[36] each of which lowers the threshold at which mood changes occur. Eventually, a mood episode can start (and become recurrent) by itself. There is evidence of hypothalamic-pituitary-adrenal axis (HPA axis) abnormalities in bipolar disorder due to stress.[37]
Other brain components which have been proposed to play a role are the mitochondria,[38] and a sodium ATPase pump,[39] causing cyclical periods of poor neuron firing (depression) and hypersensitive neuron firing (mania). This may only apply for type one, but type two apparently results from a large confluence of factors.[citation needed] Circadian rhythms and melatonin activity also seem to be altered.[40]
Environmental
Evidence suggests that environmental factors play a significant role in the development and course of bipolar disorder, and that individual psychosocial variables may interact with genetic dispositions.[32] There is fairly consistent evidence from prospective studies that recent life events and interpersonal relationships contribute to the likelihood of onsets and recurrences of bipolar mood episodes, as they do for onsets and recurrences of unipolar depression.[41] There have been repeated findings that between a third and a half of adults diagnosed with bipolar disorder report traumatic/abusive experiences in childhood, which is associated on average with earlier onset, a worse course, and more co-occurring disorders such as PTSD.[42] The total number of reported stressful events in childhood is higher in those with an adult diagnosis of bipolar spectrum disorder compared to those without, particularly events stemming from a harsh environment rather than from the child's own behavior.[43] Early experiences of adversity and conflict are likely to make subsequent developmental challenges in adolescence more difficult, and are likely a potentiating factor in those at risk of developing bipolar disorder.[44]
http://en.wikipedia.org/wiki/Bipolar_disorder
Leave it to you to post a factual filled post, thanks. I think I'm just uptight about my new job and some things that are going on there. Once that **** is handled, I'll be gravy.
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07-31-2011, 10:47 PM
#9095
Want fries with that?
iTrader: (3)
Join Date: Oct 2009
Location: Twin Cities, Minnesota
Posts: 2,011
Total Cats: 2
Did I make this too cheap?
http://minneapolis.craigslist.org/hn...523182374.html
Posted it a couple hours ago and I've had a bunch of emails on it
http://minneapolis.craigslist.org/hn...523182374.html
Posted it a couple hours ago and I've had a bunch of emails on it
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07-31-2011, 11:45 PM
#9097
Elite Member
iTrader: (2)
Join Date: Jan 2007
Location: Los Angeles, CA
Posts: 8,682
Total Cats: 130
Did I make this too cheap?
http://minneapolis.craigslist.org/hn...523182374.html
Posted it a couple hours ago and I've had a bunch of emails on it
http://minneapolis.craigslist.org/hn...523182374.html
Posted it a couple hours ago and I've had a bunch of emails on it
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